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Brain 9 , 10 (2016)
Exploring these primary pathways can provide a more comprehensive understanding of the relationship between UA and oxidative stress as well as neuroinflammation in the brain, along with the mechanism underlying the neuroprotective effects of UA in neurodegenerative diseases
It is of interest to note that a class of tertiary -chloroacetamide-containing compounds such as RSL3 has previously been shown to induce a unique iron-dependent, oxidative, non-apoptotic cell death, selectively in KRAS-mutant cells, through covalently inhibiting glutathione peroxidase 4 (refs 34, 35, 36)
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